Conţinutul numărului revistei |
Articolul precedent |
Articolul urmator |
837 16 |
Ultima descărcare din IBN: 2023-12-07 15:42 |
Căutarea după subiecte similare conform CZU |
618.3-02 (1) |
Științe medicale. Medicină (11194) |
SM ISO690:2012 PALADI, Gheorghe, STAVINSKAIA, Ludmila, TABUICA, Uliana. Infecţia ca factor etiopatogenetic în naşterea prematură. In: Buletin de Perinatologie, 2017, nr. 2(74), pp. 19-26. ISSN 1810-5289. |
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Buletin de Perinatologie | ||||||
Numărul 2(74) / 2017 / ISSN 1810-5289 | ||||||
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CZU: 618.3-02 | ||||||
Pag. 19-26 | ||||||
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Rezumat | ||||||
Premature birth is the leading cause of perinatal morbidity and mortality in the world. The effectiveness of prognosis and prevention of preterm labor remains low, despite the huge amount of research and work in this area. Premature birth can be considered as a polyethiologic syndrome with clinical manifestations as regular uterine contractions, cervical dilatation, or premature rupture of membranes. Trigger factors of preterm labor are infection of the lower pole of amniotic membranes and, as a consequence, ischemic-cervical insufficiency and premature discharge of amniotic fluid, uterine overgrowth and immune aggression. If the aforementioned etiopathogenetic factors are predicted and prevented, then the threat of premature birth will not lead to termination of pregnancy. It follows that in the delivery mechanism there can be a potentially reversible and another irreversible phase that leads to premature labor. In addition, persistent infection of the mother causes the appeaence of a syndrome of an inflammatory response in the fetus, which is the cause of severe intrauterine and neonatal morbidity, leading to cerebral palsy and other chronic pathologies or mortality. |
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